Alpha Lipioc Acid Is A Premier Anti-Oxidant In Stemulite

(THP) There is tantalizing evidence that mitochondrial decay may be a primary factor in the aging process.

Mitochondria are the chief source of endogenous oxidants, including hydrogen peroxide and the superoxide and hydroxyl radicals. This high flux of oxidants would not only be expected to damage the cell overall, but also certainly would damage the mitochondria in which the oxidants are produced.

Despite an impressive array of antioxidant defenses, such as vitamin C, glutathione and vitamin E, the level of oxidative damage to mitochondria is enormous. Experiments have shown that oxidative damage to mitochondrial DNA (the only organelle with its own DNA outside the nucleus) is 8 to 10-fold higher than the levels found in nuclear DNA and accumulates even higher levels of damage with age.

Chronic steroid use in exercise training and body building often leads to prediabetic conditions or prediabetes. One hallmark of the insulin-resistant state of prediabetes and overt type 2 diabetes is an impaired ability of insulin to activate glucose transport in skeletal muscle, due to defects in insulin receptor substrate-1 (IRS-1)-dependent signaling. An emerging body of evidence indicates that one potential factor in the etiology of skeletal muscle insulin resistance is oxidative stress, an imbalance between the cellular exposure to an oxidant stress and the cellular antioxidant defenses. Therefore, anyone who has any kind of a chronic high glucose should daily take ALA. Exposure of skeletal muscle to an oxidant stress leads to a vicious cycle of impaired insulin signaling and subsequently to reduced glucose transport activity. Numerous studies have demonstrated that treatment of insulin-resistant animals and type 2 diabetic humans with antioxidants, including alpha-lipoic acid (ALA), are associated with improvements in skeletal muscle glucose transport activity and whole-body glucose tolerance.

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